Comments by "Roger Scott Cathey" (@rogerscottcathey) on "NHS ... information" video.

  1. Yes but what allowed survival and the resistance in the first infection has not been nailed down. It is all assumed to be contact cell competency. No study of diet, no blood stream and lymphatic system assay of hydrolase enzymes titres. No, only a simplistic concept of contact cells, white blood cells, lymphocytic and phagocytic cells. The analysis of exactly what constitutes robust health and resistance to infections and cancers both has next to zero research combinatorially. The reason I think is simple: it would devolve upon adequate nutrition, something proprietary drug pushers don't want talked about too loudly . . . will not fund research for, and do not endow universities to provide education upon. Furthermore, essential nutritional elements not as yet generally recognized, though proven long ere now proven scientifically to be essential, will be continued to be ignored. One such being hydrocyanic acid. Yet we know it is essential because it is part and parcel to resistance to viruses in the respiratory epithelium of the lungs utilizing isothiocyanate, which can only be derived from HCN, hydrocyanic acid. "Hypothiocyanite produced by human and rat respiratory epithelial cells inactivates extracellular H1N2 influenza A virus" "Objective and design Our aim was to study whether an extracellular, oxidative antimicrobial mechanism inherent to tracheal epithelial cells is capable of inactivating influ­enza H1N2 virus. "Material or subjects: Epithelial cells were isolated from tracheas of male Sprague–Dawley rats. Both primary human and rat tracheobronchial epithelial cells were dif­ferentiated in air–liquid interface cultures. "Treatment A/swine/Illinois/02860/09 (swH1N2) influ­enza A virions were added to the apical side of airway cells for 1 h in the presence or absence of lactoperoxidase or thiocyanate. "Results Our data show that rat tracheobronchial epithelial cells develop a differentiated, polarized monolayer with high transepithelial electrical resistance, mucin production and expression of dual oxidases. Influenza A virions are inactivated by human and rat epithelial cells via a dual oxidase-, lactoperoxidase-and thiocyanate-dependent mechanism." Inflamm. Res. (2016) 65:71–80 DOI 10.1007/s00011-015-0892-z
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