Comments by "Terje Oseberg" (@terjeoseberg990) on "Dr. John Campbell" channel.

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  510.  @Baard2000 , That 375,000 number is reported events after vaccination. These events occur every day in people for other reasons and no reason at all. The only way to determine whether those events were caused by the vaccines is to compare the numbers to the regular background rates of these same events, and only if they see an increase will it be determined by the vaccines. (Made up number) For example if there’s are 5 heart attack per week per million men between 50 and 55 and after vaccination there are still 5, then the vaccines did not cause those 5. But if it suddenly increases to 20, then the vaccines caused 15 of them. So basically, the majority of those 375,000 cases are not caused by the vaccines, or they stop using them. They would be considered too dangerous. They’ve recently determined that Pfizer causes myocarditis is teen boys. Well, it actually doubles the rate from approximately 6 per 100,000 per year to 12 or something like that. It’s really not that many, so in the US that are still recommending that teens 12 - 17 be vaccinated. Every event after vaccination should be reported, but every event reported is not caused by the vaccines. Someone who actually understands what they are doing needs to review the numbers cases and make that determination. They are doing these reviews constantly. That’s how they discovered the blood clotting issue and the myocarditis issue. And that’s why despite there being 375,000 entries, there is nothing else there that’s concerning.
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  547.  @Campbellteaching , Methods: This observational cohort study was conducted from March to May 2020, among patients admitted to COVID-19 wards of Hospital del Mar, Barcelona, Spain. A total of 930 patients with COVID-19 were included; 92 were excluded because of previous calcifediol intake. Of the remaining 838, a total of 447 received calcifediol (532 μg on day 1 plus 266 μg on days 3, 7, 15, and 30), whereas 391 were not treated at the time of hospital admission (intention-to-treat). Of the latter, 53 patients were treated later during ICU admission and were allocated in the treated group in a second analysis. In healthy individuals, calcifediol is about 3.2-fold more potent on a weight basis than cholecalciferol. Main outcome measures were ICU admission and mortality. Results: ICU assistance was required by 102 (12.2%) participants. Out of 447 patients treated with calcifediol at admission, 20 (4.5%) required the ICU, compared to 82 (21%) out of 391 nontreated (P < .001). Logistic regression of calcifediol treatment on ICU admission, adjusted by age, sex, linearized 25-hydroxyvitamin D levels at baseline, and comorbidities showed that treated patients had a reduced risk of requiring the ICU (odds ratio [OR] 0.13; 95% CI 0.07-0.23). Overall mortality was 10%. In the intention-to-treat analysis, 21 (4.7%) out of 447 patients treated with calcifediol at admission died compared to 62 patients (15.9%) out of 391 nontreated (P = .001). Adjusted results showed a reduced mortality risk with an OR of 0.21 (95% CI, 0.10-0.43). In the second analysis, the obtained OR was 0.52 (95% CI, 0.27-0.99). Conclusion: In patients hospitalized with COVID-19, calcifediol treatment significantly reduced ICU admission and mortality.
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  1025. turbofreddy, Everyone who ends up in the ICU is there because they can’t breathe without supplemental oxygen and because they need extreme medical care, otherwise they’d be at home. Therefore, EVERYONE in the ICU is experiencing lung damage. Not just a little lung damage either. If it was just a little damage, they could stay home. They wouldn’t be in the ICU. 50% of those who are infected experience no noticeable symptoms. This doesn’t mean they are unaffected. They are, otherwise they wouldn’t be contagious. They too are experiencing lung damage. Buy it’s mild lung damage that they don’t notice, and they can most likely repair. The other 50% experience noticeable symptoms because they are suffering more damage 3/5ths of them are experiencing mild symptoms and can stay home. Some of them will suffer permanent lung damage, but not that much. They might not be able to be a top athlete any more, but they’ll be mostly OK. That’s 30% of those who are infected by the virus. The remaining 20% who are hospitalized are experiencing more severe lung damage. This damage varies, but it’s permanent. It’s scar tissue within their lungs that will be present forever so they will be left with noticeably reduced lung capacity. Many of those who end up in the ICU will require supplemental oxygen for the rest of their lives. Look up portable oxygen concentrator. These devices are manufactured for people who need supplemental oxygen just to walk around. A lot of Covid survivors will end up like this. This is not something you want to F*ck around with. I’ll risk a 1 in a million chance of dying of a blood clot rather than a 20% chance of F*cking up my lungs for life.
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  1169. Stephen Arling, One example of a bad one was the recent one with about 3,000 participants where about 2/3rd took two doses of Ivermectin 72 hours apart and the remaining 1/3rd took nothing. This study “showed” that two doses 72 hours apart is 83% effective for one month. The problem with this study is that the participants self selected. Meaning they were all offered Ivermectin, and about 1/3rd of them refused. Why this is bad, is because it’s possible that those who refused were people who believe that the virus “the flu” or a hoax or real, but not dangerous, and therefore were careless and refused to take other precautions. While those who accepted the Ivermectin might be people who are much more cautious and wore their masks diligently and maintained social distance. This is why double blind is so important. Everyone in the study need to understand that they might have received the drug, or they might not. Therefor they will all behave similarly. If they had randomly given the ivermectin to half of the 3,000 and placebo to the other half, then you’d have an even mix of both types of people in both arms of the study. So, although the study appears to show that Ivermectin is 83% effective, what it really shows is that the type of people who would choose to take Ivermectin are 83% less likely to get Covid than the type of people who would choose not to take Ivermectin. So, to determine whether a study is good or not, you have to look at the study, and if you can’t find any such problems with it, then it’s likely good. If this particular study was a double blind study with a placebo control, then it would be a great study. Unfortunately, it’s not.
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  1803.  @mayrareyes8135 , When this pandemic started, around 50% of the immunologically naive were asymptomatic upon infection, and around 30% experienced mild symptoms. Only around 10-15% required hospital care. Around 2/3 of those who required hospital care had at least 1 comorbidity, so 1/3 had no comorbidities. Since approximately 50% of the population has 1 comorbidity, those with 1 or more comorbidities are at approximately twice the risk as those with none. Therefore those with at least 1 comorbidity are at around 20% risk, and those with none are at around 10% risk of hospitalization. So, if Omicron is twice as lethal and you have no comorbidities, then your risk of hospitalization, if you were immunologically naive, was around 10%. You therefore had around a 90% chance of experiencing mild to no symptoms. However, how can you be sure you were immunologically naive? How do you know you weren’t already infected and experienced mild to no symptoms? You could have already been among the 80% who gained immunity from previous infection. Were you tested for antibodies or for a T cell response? Most of the world now has immunity either from previous infection, or vaccination. The seroprevalence in the UK is around 98%. The pandemic is pretty much over for the majority of us. The immunologically naive are now extremely rare, and most of the unvaccinated, without getting tested, have no clue whether they are immunologically naive or not. You got lucky, and you are now no longer immunologically naive. However, you have no clue what damage this infection may have done to your body, and what the long term consequences might be. There have been no long term studies, and the short term studies don’t look good. How SARS-CoV-2 Causes Brain, Kidney, Pancreas, & Heart Damage https://youtu.be/g9Ftwk4loPA
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  2165. Chris H, Here’s how PCR works in really simple terms that hopefully your relative can understand. They take a sample from your nose, and put it in a liquid that preserves RNA. Then they add the materials needed to duplicate the RNA the same way it duplicates inside your cells. Once they’ve all been copied, the copies are attached to the originals. When they heat the sample, the copies separate from the originals and you now have twice as many bits of RNA. Then they let it cool down, and they start copying again. The cycles are just the heating and cooling cycle, and you double the number of copies with every cycle. There’s more to it like there’s a mechanism so only the bits of RNA that match a pattern are copied. The cycle count is just a measure of how much RNA was in the sample. But how the sample was taken also influences how much RNA was present in the sample. Requiring 35 cycles for a positive result vs 40 definitely changes the number of positive results, but with 40, the RNA fragments were there. Why? If you were never infected, why would you have SARS-CoV-2 RNA fragments in your sample? It’s a good thing that the sample contained so few RNA fragments that they needed 40 cycles to detect them. It means that you’re probably not contagious. Maybe you got infected by the virus, but your body fought it off. Or maybe you just got it and you haven’t gotten sick yet. If I were a doctor and someone tested positive with a CT of 40, I’d immediately prescribe Ivermectin and I’d test their blood for Vitamin D and if it’s low, I’d prescribe Calcifediol (a form of Vitamin D that’s absorbed immediately). Then I’d explain that they either just got infected, or they’ve been infected and they’re getting over it already. And I’d test them again in a week to see if the RNA is increasing or decreasing. It might also be worth testing their blood for antibodies. If antibodies are present it means they’ve been infected for a while and are probably getting over it.
    1
  2166. Chris H, For PCR testing to detect only SARS-CoV-2, they have to analyze the RNA for all coronaviruses and choose to amplify only a segment that exists in SARS-CoV-2. Sure, there are many similarities between coronaviruses. But they’re not identical, or they would be the same virus. So there are obviously differences. And sure, There’s no way to tell from the PCR test whether the sample contains full viruses, or only fragments. But isn’t it obvious to you that if you have a whole bunch of virus RNA in your nose, that your body is manufacturing them? Otherwise, where are they coming from? Do you really believe you could have RNA fragments sitting in your nose a month after your body stopped manufacturing them? Your body isn’t constantly excreting mucus and sending immune cells there to kill bacteria and clean up the mess? I doubt RNA fragments could last a few hours in there before they are somehow removed by some mechanism or another. This is all part of the innate immune system that’s supposed to keep viruses out of our bodies. They’re are millions of bacteria in the air that we’re breathing in constantly, our bodies are constantly dealing with them to keep them out. These RNA fragments are mixed with these bacteria and they are consumed by our innate immune system along with them. Anyone who believes that an RNA fragment is going to last very long up there is nuts. Unless there reproducing faster than they can be destroyed, they’re going to be gone. So basically, if they’re detected, There’s viral replication going on in your body. Viral replication means there are fully intact viruses somewhere in your body. Whether they are in your nose or not is unknown from these test results, but most likely there are. But this is only a question of whether you’re contagious or not, NOT whether you’re infected or not. And if we don’t know whether you’re contagious or not, then just behave like you are, and medicate you like you just got it and could get worse until they’ve proven otherwise with a negative test result. There’s no point taking any chances.
    1
  2167. Sharky Moon, What are you going to do when Pfizer gets full FDA approved next week? When it’s no longer approve for emergency use, but it’s fully approved? What then? Your whole argument that “it’s not approved” goes down the drain. This is going to crush your whole world view. I hope you don’t go into mental meltdown or something. You should go see your therapist and get your meds in advance, because full FDA approval is inevitable, because the Pfizer vaccine is safe and effective. Moderna, J&J, AstraZeneca, Novavax will also be fully FDA approved eventually too, because they’re all safe and effective. It’s pretty obvious bro. They vaccinated over 15,000 in the phase 3 trials, and they didn’t even start the phase 3 trial until after they proved that the vaccines were safe in the phase 1 (human safety trials). They’re not going to let them vaccinate 15,000 people with a vaccine that might not be safe. That’d be dumb. If 10% of them died, nobody would take the vaccine. They’d be wasting their time and money. They’d be stupid to do that. So obviously they’re going to prove that it’s safe BEFORE they invest a dime in a phase 3 trial. Isn’t that obvious? And after vaccinating 15,000 people in the phase 3 trial, not one of them died because of the vaccine. And now, over 1.4 billion people have been vaccinated. If 0.1% died, there would be 1.4 million dead people from the vaccines. You don’t think we’d notice that? The antivaxxer a are making a big deal out of 4,000 people who probably would have died already without the vaccine. To really know whether the vaccines caused it or not, we need to compare those 4,000 people with how many die without vaccines. Did the vaccines cause an increase in deaths or not? If there’s an increase, then it’s probably the vaccines. If there’s no increase, then it’s not the vaccines. So, is there an increase in deaths or not? Don’t trust what I tell you. Go look it up. Go check to see if the vaccines are causing deaths to increase or not. If the vaccines are killing people there will be an increase. Right? So you go find your evidence and then present it to people. Or, go find the evidence that you’re wrong. I already know what you’re going to find.
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  2341.  @solobano570 , This is a much better study that actually proves something. Methods: This observational cohort study was conducted from March to May 2020, among patients admitted to COVID-19 wards of Hospital del Mar, Barcelona, Spain. A total of 930 patients with COVID-19 were included; 92 were excluded because of previous calcifediol intake. Of the remaining 838, a total of 447 received calcifediol (532 μg on day 1 plus 266 μg on days 3, 7, 15, and 30), whereas 391 were not treated at the time of hospital admission (intention-to-treat). Of the latter, 53 patients were treated later during ICU admission and were allocated in the treated group in a second analysis. In healthy individuals, calcifediol is about 3.2-fold more potent on a weight basis than cholecalciferol. Main outcome measures were ICU admission and mortality. Results: ICU assistance was required by 102 (12.2%) participants. Out of 447 patients treated with calcifediol at admission, 20 (4.5%) required the ICU, compared to 82 (21%) out of 391 nontreated (P < .001). Logistic regression of calcifediol treatment on ICU admission, adjusted by age, sex, linearized 25-hydroxyvitamin D levels at baseline, and comorbidities showed that treated patients had a reduced risk of requiring the ICU (odds ratio [OR] 0.13; 95% CI 0.07-0.23). Overall mortality was 10%. In the intention-to-treat analysis, 21 (4.7%) out of 447 patients treated with calcifediol at admission died compared to 62 patients (15.9%) out of 391 nontreated (P = .001). Adjusted results showed a reduced mortality risk with an OR of 0.21 (95% CI, 0.10-0.43). In the second analysis, the obtained OR was 0.52 (95% CI, 0.27-0.99). Conclusion: In patients hospitalized with COVID-19, calcifediol treatment significantly reduced ICU admission and mortality.
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  2684. asearchie, Ivermectin is not a “non-pharmaceutical treatment” though. The issue here is to we need an RCT, and the FLCCC should just do one. Obviously big pharma will never do it. Duhhh.... Why would they? All I’m saying it’s that FLCCC should do it. The medication is cheap. I heard that it’s $168/kg. A single dose is measured in micrograms. An RCT should not be expensive or difficult. You’re doing exactly what everyone else is doing, including Tess Lawrie. Why? Just do the damned study already. I’ve been saying this same thing for months, and yet it still hasn’t happened. I’m starting to believe they won’t do it because maybe they know it doesn’t work. Who the hell knows? If a thousand people do a thousand small studies with 20-30 patients each, do you believe the ones that fail will be published? Why would they published all the failed studies? They’re obviously only going to publish the successful ones, right? Maybe that’s what’s happening and we’re only seeing the studies that show positive results. How the hell would we know? We wouldn’t. So basically, maybe the sh!t works, or maybe it doesn’t. You can go ahead and look at their website and believe it works, but I want to see proof. So do a bunch of doctors, scientists, and policy makers. It makes sense. I have an actionable item here, and what do you have? You have, “but, but, but, look at the meta study”. Well, garbage in = garbage out. They want an RCT, so do an RCT. It’s that simple. If it works, the RCT will show positive results. I hope the FLCCC reads my comments and does it. Just get it over with.
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  3224. Stephen Arling, Methods A prospective cohort study was conducted at AIIMS Bhubaneswar, which provides both COVID and Non-COVID care since March 2020. All employees and students of the institute who provided written informed consent participated in the study.Uptake of two-doses of oral ivermectin (300 μg/kg at a gap of 72 hours) was considered as exposure. The primary outcome of the study was COVID-19 infection in the following month of ivermectin consumption diagnosed by RTPCR as per Government of India testing criteria guidelines.The log-binomial model was used to estimate adjusted relative risk, and the Kaplan- Meier failure plot was used to estimate the probability of COVID-19 infection with follow-up time. Results Of 3892 employees, 3532 (90.8%) participated in the study. The ivermectin uptake was 62.5% and 5.3% for two-doses and single-dose, respectively. Participants who took ivermectin prophylaxis had a lower risk of getting symptoms suggestive of SARS-CoV-2 infection(6% vs 15%). HCWs who had taken two-doses of oral ivermectin have a signi cantly lower risk of contracting COVID-19 disease during the following month (ARR 0.17; 95% CI, 0.12-0.23). Females had a lower risk of contracting COVID-19 than males (ARR 0.70 95% CI, 0.52-0.93). The absolute risk reduction of SARS-CoV-2 infection was 9.7%. Only 1.8% of the participants reported adverse events, which were mild and self-limiting. Conclusion and relevance Two-doses of oral ivermectin (300 μg/kg given 72 hours apart) as chemoprophylaxis among HCWs reduces the risk of COVID-19 infection by 83% in the following month. Safe, effective, and low-cost chemoprophylaxis have relevance in the containment of pandemic alongside vaccine. https://assets.researchsquare.com/files/rs-208785/v1/d6ff79a3-d354-4aba-a6b0-4bc123bbd225.pdf
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  3341. microcolonel, They tested the vaccines in no human primates first to verify three things. 1. The vaccines don’t kill the primates or cause severe adverse reactions. 2. The vaccines stimulate the production of the same antibodies in non human primates that they believes were the active antibodies in humans who were previously infected with SARS-CoV-2. 3. And that the vaccines actually made the non human primates immune to the virus. The experiments proved all 3. They then presented their results to the FDA to get approval to conduct human safety trials. In this trial they proved two things. 1. No humans suffered severe adverse reactions or died from the vaccine. 2. The vaccines caused human immune cells to make the same antibodies that the non human primates made which were the same ones made by those who have been infected by the virus. Then AFTER presenting THIS data to the FDA, they got permission to conduct human efficacy trials. (This is simple sh!t bro. It’s how scientific research is conducted.) So, they had a phase 3 human efficacy trial with over 30,000 participants where half got the vaccine and the other half got a placebo or whatever (not the experimental vaccine). Then, only after the results of the phase 3 human efficacy trial proved that the vaccine were effective, did the FDA approve use in the general public. These vaccines have been proven beyond a shadow doubt to be extremely safe and extremely effective. If you are unaware of this, you have clearly not looked into it, or you have been reading garbage on antivaxxer websites. Unfortunately that’s what many of these people are doing. These antivaxxer promoters have brainwashed millions of sheeple.
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